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Biology Predictions/Thoughts (1 Viewer)

aqwerty13402

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Besides the inevitable (A kidney short answer), what are we expecting this year. I think the 100%'s are:

1. Kidney short answer
2. Some evaluation of epidemiology
3. Some variation of polypeptide synthesis (likely attatched to mutation)
4. Biotechnology (i'm hoping / predicting gene cloning or whole organism cloning to pop up)
5. Pedigree / inheretence. - also feeling like we might get one of the weird things like incomplete dominance, co-dominance etc
6. A FAT feedback loop Q. (HOPEFULLY GLUCOSE)

there's probably other staples but i cant remember off the top of my head.
 

roseviolett

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some more probable ones
- something to do with 5.5 and talking about a project used to collect population genetics information
- how a named australian plant responds to a named pathogen
- bush medicine used by Aboriginal peoples, maybe a question on intellectual property of Aboriginal peoples' medicine
- comparing forms of reproduction advantages/something in the multiple choice about advantages
 

aqwerty13402

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some more probable ones
- something to do with 5.5 and talking about a project used to collect population genetics information
- how a named australian plant responds to a named pathogen
- bush medicine used by Aboriginal peoples, maybe a question on intellectual property of Aboriginal peoples' medicine
- comparing forms of reproduction advantages/something in the multiple choice about advantages
OMG I HAVE PREPARED THE AUSTRALIAN PLANT THING LMAOOO.

AUSTRO PUCINAPSIDII OR SMTHG
 

xoNat

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Besides the inevitable (A kidney short answer), what are we expecting this year. I think the 100%'s are:

1. Kidney short answer
2. Some evaluation of epidemiology
3. Some variation of polypeptide synthesis (likely attatched to mutation)
4. Biotechnology (i'm hoping / predicting gene cloning or whole organism cloning to pop up)
5. Pedigree / inheretence. - also feeling like we might get one of the weird things like incomplete dominance, co-dominance etc
6. A FAT feedback loop Q. (HOPEFULLY GLUCOSE)

there's probably other staples but i cant remember off the top of my head.
8 marker on transcription and translation with a required diagram
 

roseviolett

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wait could u send notes on this? it sounds a lot easier to remember HAHA
yeah sure!
Crown rot in Eucalyptus
Fungal pathogen: Phytophthora Cinnamomi - travels in water
  • Physical/Innate:
    • Thick waxy cuticles on leaves, thick bark and dry leaf surface prevent entry of pathogens.
    • Vertical hanging of leaves prevents water from collecting on the leaf surface
    • Closing stomata
  • Chemical barriers: stored oils (eucalyptus oil) inside plant tissues act as antifungal agents to destroy the pathogen.
    • Saponins are present all over the bark and inhibit the entry of fungal cells. Tannins are also present in their bark which are toxic to fungal cells.
  • Active response:
    • Thickening of the cell walls prevents the spread to other cells.
    • protective tissue forms beneath the infected tissue to separate the infected tissue from healthy tissue and prevent spread of disease.
    • Secretion of protective gums seal the infected area, creating a physical barrier
    • Rapid cell death (apoptosis) stops the infection from spreading by sealing off the area or forming a gall. This is due to the R-genes (resistance genes) present in eucalyptus tissues which respond to chemicals released by the pathogen and initiate cell death.
 

aqwerty13402

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yeah sure!
Crown rot in Eucalyptus
Fungal pathogen: Phytophthora Cinnamomi - travels in water
  • Physical/Innate:
    • Thick waxy cuticles on leaves, thick bark and dry leaf surface prevent entry of pathogens.
    • Vertical hanging of leaves prevents water from collecting on the leaf surface
    • Closing stomata
  • Chemical barriers: stored oils (eucalyptus oil) inside plant tissues act as antifungal agents to destroy the pathogen.
    • Saponins are present all over the bark and inhibit the entry of fungal cells. Tannins are also present in their bark which are toxic to fungal cells.
  • Active response:
    • Thickening of the cell walls prevents the spread to other cells.
    • protective tissue forms beneath the infected tissue to separate the infected tissue from healthy tissue and prevent spread of disease.
    • Secretion of protective gums seal the infected area, creating a physical barrier
    • Rapid cell death (apoptosis) stops the infection from spreading by sealing off the area or forming a gall. This is due to the R-genes (resistance genes) present in eucalyptus tissues which respond to chemicals released by the pathogen and initiate cell death.
thank uuuu.

yeah i havent seen this in a response (or even plant immune response) as a short answer q in a while.
 

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